Effect of Cushing’s syndrome on the individual
Cortisol is involved in the regulation of blood sugar, metabolism, and inflammation. Much of its exposure causes Cushing’s syndrome, which has adversely affected the individual by developing hypertension and type 2 diabetes.
The development of hypertension and diabetes, and their potential complications on the patient.
In this case, the development of hypertension and diabetes stems from the use of Glucocorticoid medications. Glucocorticoids increases blood glucose production in the liver and reduce the sensitivity of the cells to insulin (Hodgens & Sharman, 2021). Higher exposure to this medication causes excess production of glucose, which then accumulates in the blood, causing a rise in the blood sugar level, thereby leading to diabetes. Similarly, excess glucocorticoids cause high blood pressure which may lead to hypertension. Other potential complications include absorption of calcium which leads to osteoporosis and increase cholesterol and triglyceride levels among others.
Potential problems that Mrs. A may experience from long-term use of glucocorticoids
From the long-term use of glucocorticoids, Mrs. A may suffer a loss of muscle tissue bringing with it other complications such as weight gain, weakened bones, slow healing, irregular menstrual cycle, and depression.
Reference
Hodgens, A., & Sharman, T. (2021). Corticosteroids. In StatPearls [Internet]. StatPearls Publishing. Web.